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Human Nav1.5 F1486 deletion associated with long-QT syndrome leads to deficiency in inactivation and reduces lidocaine sensitivity

رسائل ماجستير ،رسائل دكتوراه في الصيدلة.pdf ( 481 ) :: Human Nav1.5 F1486 deletion associated with long-QT syndrome leads to deficiency in inactivation and reduces lidocaine sensitivity رسالة دكتوراه - تحميل مباشر

تفاصيل كتاب Human Nav1.5 F1486 deletion associated with long-QT syndrome leads to deficiency in inactivation and reduces lidocaine sensitivity

التصنيف: رسائل ماجستير ودكتوراه -> الصيدلة

حجم الملف: 2,589 KB
نوع الملف: pdf
أضيف بواسطة: Y4$$3R N3T
بتاريخ: 19-08-2018
عدد مرات التحميل: 1
مرات الزيارة: 330

عرض جميع الكتب التي أضيفت بواسطة: Y4$$3R N3T

تحميل كتاب Human Nav1.5 F1486 deletion associated with long-QT syndrome leads to deficiency in inactivation and reduces lidocaine sensitivity

« GCN5-B is a Novel Nuclear Histone Acetyltransferase that is Crucial for Viability in the Protozoan Parasite Toxoplasma gondii

CONTRIBUTIONS OF TM5, ECL3 AND TM6 OF HUMAN BCRP TO ITS OLIGOMERIZATION ACTIVITIES AND TRANSPORT FUNCTIONS »

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Human Nav1.5 F1486 deletion associated with long-QT syndrome leads to deficiency in inactivation and reduces lidocaine sensitivity

تفاصيل كتاب Human Nav1.5 F1486 deletion associated with long-QT syndrome leads to deficiency in inactivation and reduces lidocaine sensitivity

« GCN5-B is a Novel Nuclear Histone Acetyltransferase that is Crucial for Viability in the Protozoan Parasite Toxoplasma gondii

CONTRIBUTIONS OF TM5, ECL3 AND TM6 OF HUMAN BCRP TO ITS OLIGOMERIZATION ACTIVITIES AND TRANSPORT FUNCTIONS »

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Human Nav1.5 F1486 deletion associated with long-QT syndrome leads to deficiency in inactivation and reduces lidocaine sensitivity

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